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Inhibition of AMPK through Lyn-Syk-Akt enhances FcεRI signal pathways for allergic response
Kai Chun Lin, Duen Yi Huang, De Wei Huang, Shiang Jong Tzeng, Wan Wan Lin
醫學科學研究所
藥理學科
研究成果
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引文 斯高帕斯(Scopus)
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Immunology and Microbiology
Immunoglobulin E
100%
Hypersensitive Response
100%
Passive Skin Anaphylaxis
100%
Tumor Necrosis Factor
50%
Mast Cell
50%
Mouse Model
50%
Blood Vessel Permeability
50%
Akt Signaling
50%
Gene Silencing
50%
Pharmacology, Toxicology and Pharmaceutical Science
Allergic Reaction
100%
Metformin
100%
Passive Skin Anaphylaxis
66%
Immunoglobulin E
66%
Tumor Necrosis Factor
33%
Mouse Model
33%
Phosphotransferase
33%
Receptor
33%
Protein Tyrosine Kinase
33%
6,7 Dihydro 4 Hydroxy 3 (2' Hydroxy 1,1' Biphenyl 4 Yl) 6 oxothieno[2,3 B]pyridine 5 Carbonitrile
33%
5 Amino 4 Imidazolecarboxamide Riboside
33%
Disease
33%
Medicine and Dentistry
Cell Signaling Pathway
100%
AMP-Activated Protein Kinase
100%
Allergic Response
100%
Glycon
17%
Passive Skin Anaphylaxis
11%
Immunoglobulin E
11%
In Vitro
5%
Disease
5%
Tumor Necrosis Factor
5%
Receptor
5%
Protein Tyrosine Kinase
5%
Akt Signaling
5%
Blood Vessel Permeability
5%
Kinase Assay
5%
5 Amino 4 Imidazolecarboxamide Riboside
5%
Gene Silencing
5%
6,7 Dihydro 4 Hydroxy 3 (2' Hydroxy 1,1' Biphenyl 4 Yl) 6 oxothieno[2,3 B]pyridine 5 Carbonitrile
5%
Mast Cell
5%
Biochemistry, Genetics and Molecular Biology
Syk
100%
AMP-activated Protein Kinase
100%
Immunoglobulin E
11%
Mouse Model
5%
Gene Silencing
5%
Akt Signaling
5%
Blood Vessel Permeability
5%
Src Family Kinase
5%
Kinase
5%
Phosphotransferase
5%
Eicosanoid Receptor
5%
Tumor Necrosis Factor
5%
Mast Cell
5%