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Behavioral stress modifies hippocampal synaptic plasticity through corticosterone-induced sustained extracellular signal-regulated kinase/mitogen-activated protein kinase activation
Chih Hao Yang
, Chiung Chun Huang
, Kuei Sen Hsu
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雜誌貢獻
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152
引文 斯高帕斯(Scopus)
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Neuroscience
Extracellular Signal-Regulated Kinases
100%
Mitogen-Activated Protein Kinase
100%
Synaptic Plasticity
100%
Corticosterone
100%
Long-Term Potentiation
66%
Long-Term Depression
66%
Mitogen Activated Protein Kinase 3
66%
Brain-Derived Neurotrophic Factor
33%
Western Blot
33%
Receptor Antagonist
33%
Kinase
33%
Receptor Tyrosine Kinase
33%
Tyrosine Kinase
33%
Protein Kinase C
33%
Glucocorticoid Receptor
33%
Hyperphosphorylation
33%
Antisense Oligonucleotide
33%
U0126
33%
Estrone
33%
Ras Protein
33%
K252a
33%
Bisindolylmaleimide
33%
Pharmacology, Toxicology and Pharmaceutical Science
Mitogen Activated Protein Kinase 3
100%
Mitogen Activated Protein Kinase
100%
Mitogen Activated Protein Kinase Kinase
100%
Corticosterone
100%
Western Blot
50%
Phosphotransferase
50%
1,4 Diamino 1,4 Bis(2 Aminophenylthio) 2,3 Dicyanobutadiene
50%
Protein Tyrosine Kinase
50%
K 252a
50%
Glucocorticoid Receptor Antagonist
50%
Antisense Oligonucleotide
50%
Brain Derived Neurotrophic Factor
50%
Ras Protein
50%
Protein Kinase C
50%
Bisindolylmaleimide
50%
Estrone
50%
Biochemistry, Genetics and Molecular Biology
Extracellular Signal-Regulated Kinases
100%
Corticosterone
100%
Nerve Cell Plasticity
100%
Behavioral Stress
100%
Mitogen-Activated Protein Kinase
100%
Long Term Potentiation
50%
Long Term Depression
50%
Western Blot
25%
Tyrosine Kinase
25%
Oligonucleotide
25%
Receptor Antagonist
25%
Hyperphosphorylation
25%
Small GTPase
25%
Antisense
25%
U0126
25%
Kinase
25%
Receptor Tyrosine Kinase
25%
Phosphotransferase
25%
Protein Kinase C
25%
Brain-Derived Neurotrophic Factor
25%
Glucocorticoid Receptor
25%
Bisindolylmaleimide
25%