摘要
Background: Despite a clear delineation of atrioventricular (AV) node and
perinodal inputs within the Koch's triangle, the anatomical mechanism underlying the occurrence of AV nodal reentrant tachycardia (AVNRT) in human
remains unknown. Methods: We hypothesized that the geometrical interaction between the AV junctional structures including the Koch's triangle, coronary sinus (CS) and AV node (indicated by AV nodal artery ending) determines the occurrence of AVNRT in human, Anatomy assessment via orthogona[ selective coronary arteriography and simultaneous CS venography plus right ventriculography were performed and compared between 57 patients (pts) with AVNRT (group A, 20 men, 37 women, age 54:1:11 years) and 28 pts without (group B, 13 men, 15 women, age 48 ~ 7 years), Results: Anatomical characteristics of the Koch's triangle (A), the CS orifice (CSO; and the location of the AV node in group A and B pts were listed as follows.
perinodal inputs within the Koch's triangle, the anatomical mechanism underlying the occurrence of AV nodal reentrant tachycardia (AVNRT) in human
remains unknown. Methods: We hypothesized that the geometrical interaction between the AV junctional structures including the Koch's triangle, coronary sinus (CS) and AV node (indicated by AV nodal artery ending) determines the occurrence of AVNRT in human, Anatomy assessment via orthogona[ selective coronary arteriography and simultaneous CS venography plus right ventriculography were performed and compared between 57 patients (pts) with AVNRT (group A, 20 men, 37 women, age 54:1:11 years) and 28 pts without (group B, 13 men, 15 women, age 48 ~ 7 years), Results: Anatomical characteristics of the Koch's triangle (A), the CS orifice (CSO; and the location of the AV node in group A and B pts were listed as follows.
| 原文 | 英語 |
|---|---|
| 出版狀態 | 已發佈 - 2月 1998 |
| 對外發佈 | 是 |