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The hyperaggregability of platelets from normal pregnancy is mediated through thromboxane A
2
and cyclic AMP pathways
J. R. Sheu
,
G. Hsiao
, M. Y. Shen
, W. Y. Lin
, C. R. Tzeng
Graduate Institute of Medical Sciences
Department of Pharmacology
The Ph.D. Program for Translational Medicine
Ph.D. Program in Medical Neuroscience
TMU Research Center of Neuroscience
Ph.D. Program in Drug Discovery and Development Industry
Ph. D. Program in the Clinical Drug Development of Herbal Medicine
Department of Ophthalmology
TMU Research Center of Cell Therapy and Regeneration Medicine
Department of Obstetrics and Gynecology
Taipei Medical University Hospital
Research output
:
Contribution to journal
›
Article
›
peer-review
17
Citations (Scopus)
Overview
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Dive into the research topics of 'The hyperaggregability of platelets from normal pregnancy is mediated through thromboxane A
2
and cyclic AMP pathways'. Together they form a unique fingerprint.
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Pharmacology, Toxicology and Pharmaceutical Science
Cyclic AMP
100%
Thrombin
100%
Thromboxane A2
100%
Fluorescent Dye
20%
Indometacin
20%
Fluorescein Isothiocyanate
20%
Fibrinogen Receptor
20%
Triflavin
20%
Sodium Ion
20%
Fura 2 Acetoxymethyl Ester
20%
Preeclampsia
20%
Adenosine Diphosphate
20%
Imidazole
20%
Prostaglandin E1
20%
Immunology and Microbiology
Platelet
100%
Thromboxane A2
100%
Thrombin
71%
Thrombocyte Activation
28%
Thrombocyte Aggregation
28%
Cell pH
28%
Agonist
14%
Pregnancy
14%
Platelet Reactivity
14%
Eclampsia
14%
Fibrinogen Receptor
14%
Thrombocyte Function
14%
Prostaglandin E1
14%