Glutamine administration ameliorates sepsis-induced kidney injury by downregulating the high-mobility group box protein-1-mediated pathway in mice

Medicine and Dentistry

• Sepsis 100%
• Kidney Injury 100%
• High Mobility Group B1 Protein 100%
• Glutamine 100%
• Acute Kidney Injury 37%
• Puncture 37%
• Ligation 37%
• Sodium Chloride 37%
• Protein Expression 25%
• Receptor 25%
• Toll Like Receptor 4 25%
• Mediator 25%
• Advanced Glycation End-Product 25%
• Antioxidant 12%
• Oxidative Stress 12%
• Anti-Inflammatory Drug 12%
• Single Drug Dose 12%
• Tail Vein 12%
• Myeloid Differentiation 12%
• 3 Nitrotyrosine 12%
• Kidney Tissue 12%

Pharmacology, Toxicology and Pharmaceutical Science

• Mouse 100%
• Kidney Injury 100%
• High Mobility Group B1 Protein 100%
• Sepsis 100%
• Glutamine 100%
• Acute Kidney Failure 37%
• Sodium Chloride 37%
• Advanced Glycation End Product 25%
• Receptor 25%
• Toll Like Receptor 4 25%
• Anti-Inflammatory Drug 12%
• 3 Nitrotyrosine 12%
• Synaptotagmin I 12%
• Inflammation 12%
• Antioxidant 12%

Immunology and Microbiology

• Mouse 100%
• Protein Expression 40%
• TLR4 40%
• Mediator 40%
• Glycation 40%
• Vein 20%
• Systemic Inflammation 20%
• Kidney Tissue 20%
• Myeloid Differentiation 20%
• Oxidative Stress 20%