β-Endorphin omission analogs: Dissociation of immunoreactivity from other biological activities

C. H. Li, D. Yamashiro, L. Tseng, W. C. Chang, P. Ferrara

Research output: Contribution to journalArticlepeer-review

17 Citations (Scopus)

Abstract

An analog of human β-endorphin with omission of four residues at positions 11, 14, 20, and 22 has been synthesized. This analog and other synthetic analogs with deletion of a single amino acid at position 2, 5, 6, 10, 11, 12, 13, 15, or 22 have been assayed for analgesic potency, ileal opiate activity, opiate receptor-binding activity, and immunoreactivity. Results show that deletion of a single amino acid of the β-endorphin molecule outside of the enkephalin segment to give (des-Gln11-, des-Thr:12-, des-Pro13-, des-Leu14-, des-Val15-, des-Asn20-, des- Ile22-β-endorphin) markedly reduced or abolished the immunoreactivity yet gave substantial retention of opiate potencies. Deletion of a single amino acid of β-endorphin within the enkephalin segment (des-Gly2- or des-Met5-β-endorphin) did not markedly affect the immunoactivity; however, the opiate activities were abolished or markedly reduced. The data indicate a clear dissociation of immunoactivity from analgesic, ileal-opiate, and opiate receptor-binding activities.

Original languageEnglish
Pages (from-to)3211-3214
Number of pages4
JournalProceedings of the National Academy of Sciences of the United States of America
Volume77
Issue number6 I
DOIs
Publication statusPublished - Dec 1 1980
Externally publishedYes

ASJC Scopus subject areas

  • General

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