Could Bicalutamide–Induced Renal Fibrosis Take the Pathway Tgf-B1/Wnt That Further Triggers Mesangial Mitochondrial Dysfunction and Metabolic Reprogramming? - Evaluation of Prognosis on Cotherapy of Nac, Nad+, Riboflavin, and Coq?

Project: A - Government Institutionb - National Science and Technology Council

Project Details

StatusFinished
Effective start/end date8/1/197/31/20

Keywords

  • diabetic renal fibrosis
  • mitochondria dysfunction
  • tissue hypoxia
  • antiandrogen
  • metabolic reprogramming